Lymphoma Abstract Award Recipients

2018 Recipients

Elena Battistello
École Polytechnique Fédérale de Lausanne
Lausanne, Switzerland
Multimodal Inhibition of BCR Signaling in DLBCL Impacts DLBCL Evolution
Joanne Dai
Duke University
Durham, NC
Epstein-Barr Virus Latent Infection of B Cells Promotes Germinal Center-Like Apoptosis Resistance
Franziska Gehringer
Institute of Physiological Chemistry
Ulm, Germany
Tight Regulation of the PI3K-AKT-FOXO1 Axis Is Essential for Maintenance of the Dark Zone Program and Survival of Burkitt Lymphoma
James Heward
Barts Cancer Institute, Queen Mary University of London
London, United Kingdom
KDM5 Inhibition Is A Novel Therapeutic Strategy for the Treatment of KMT2D Mutant Lymphomas
Kallesh Jayappa
Beirne B. Carter Center for Immunology Research, University of Virginia
Charlottesville, VA
Diverse Microenvironmental Agonists Generate Drug Tolerant Persister Phenotype and Cancer Cell Proliferation in B Cell Malignancies
Michael Jin
Stanford University Medical Center
Stanford, CA
Circulating Tumor DNA as a Biomarker for the Noninvasive Genotyping and Monitoring of Classical Hodgkin Lymphoma
Masaharu Kawashima
Tokai University, Isehara-shi
Kanagawa, Japan
Upregulation of Immune Checkpoint Markers PDL1/2 on Monocytes Induced by Direct Contact With Hodgkin Cells
Gero Knittel
University Hospital Cologne
Cologne, Germany
MYD88 p.L265P and BCL2 Overexpression Cooperate in Lymphomagenesis in Mice
Marvyn Koning
Leiden University Medical Center
Leiden, Netherlands
Primary Cutaneous Follicle Center Lymphomas Express Heavily Mutated B-Cell Receptors With Acquired N-Glycosylation Motifs and Lack Ongoing Somatic Hypermutation
Olga Kutovaya
BC Cancer
Vancouver, Canada
UBR5 Mutations in Mantle Cell Lymphoma Lead to Increased Proliferation Through A Cyclin D1 Dependent Mechanism
Marion Lacroix
IRCM (Clinical Research Institute of Montreal)
Montreal, Canada
The Role of the RNA Helicase DDX3X in Human Burkitt Lymphoma
Anna Lollies
Institute of Cell Biology (Cancer Research)
Essen, Germany
Molecular Mechanisms in the Pathogenesis of Composite Lymphomas
Kathryn Lurain
National Cancer Institute, National Institutes of Health
Washington, DC
Radiation-Sparing Treatment of HIV-Related Primary Central Nervous System Lymphoma With Antiretroviral Therapy, Rituximab, and High-Dose Methotrexate
Sayeef Mirza
University of South Florida
Tampa, FL
Genomic Landscapes of CTCL and Sezary Syndrome
Soumya Sundara Rajan
University of Miami
Miami, FL
Genomic Editing of Transplantable Hematopoietic Stem Cells With CRISPR/Cas9 to Generate Npm1-Alk Through Chromosomal Translocation Accurately Models ALK+ T-Cell Lymphoma in Mice
Ariel Sindel
VCU Health System
Richmond, VA
Role of the PI3K Pathway in the Pathogenesis of Marginal Zone Lymphoma
Vipul Shukla
La Jolla Institute for Allergy and Immunology
La Jolla, CA
Tet Proteins Regulate Enhancer Function and Class Switch Recombination During B Cell Activation
Ashima Shukla
Sanford Burnham Prebys Medical Discovery Institute
San Diego, CA
Change in B Cell Receptor Isotype Alters the Tumor Forming Properties of Lymphoma B Cells
Richard Sora
Northwestern University Feinberg School of Medicine
Glenview, IL
Blocking Two Pathways of P27Kip1Degradation Delays Tumor Onset in Mouse Model of Myc-driven Lymphoma
Yasuhito Suehara
University of Tsukuba
Tsukuba, Japan
Tp53 mutations Found in the Serum of Malignant Lymphoma Patients at Remission Derive From Clonal Hematopoiesis Rather Than Minimal Residual Disease
Justin Taylor
Memorial Sloan Kettering Cancer Center
New York, NY
Oncogenic Mutations in XPO1 Promote Lymphoid Transformation by Altering Nuclear-Cytoplasmic Localization of NFκB Signaling Intermediates
Elena Viganò
BC Cancer Research Centre
Vancouver, Canada
Recurrent IL4R Somatic Mutations in Diffuse Large B-Cell Lymphoma Lead to an Altered Gene Expression Profile and Changes in Tumor Microenvironment Composition
Jillian Wise
Oslo University Hospital, Institute for Cancer Research
Westport , MA
Relapsed/refractory Diffuse Large B-Cell Lymphomas Demonstrate Late Divergent Evolution and Increased TP53 Network Aberrations
Julieta Sepulveda-Yanez
Leiden University Medical Center
Leiden, Netherlands
Differential Mutational Pattern in Indolent B-Cell Lymphomas
Tetsumi Yoshida
Hiroshima University
Hiroshima, Japan
Patterns of IgH Rearrangement Can Predict Prognosis of Diffuse Large B-Cell Lymphoma in Association With CDC25A Overexpression

2016 Recipients

Idit Sagiv-Barfi
Stanford University
Stanford, CA
Which Checkpoint Antibody Combinations Are Synergistic?
Emilia Bialopiotrowicz, PhD
Institute of Hematology and Transfusion Medicine
Warsaw, Poland
Functional Link Between Heat Shock Protein HSP90alpha and Sirtuin 1 (SIRT1) in the Pathogenesis of Diffuse Large B-Cell Lymphoma
Rebecca Boddicker, PhD
Mayo Clinic
Rochester, MN
Integrated Mate-Pair and RNA Sequencing Identifies Novel, Targetable Gene Fusions in Peripheral T-Cell Lymphoma
Alexander Craig, MPhil
Stanford University Medical Center
Stanford, CA
Methods of Circulating Tumor DNA Quantification in Plasma
Ondrej Havranek, MD, PhD
The University of Texas MD Anderson Cancer Center
Houston, TX
Features of Tonic B-Cell-Receptor Signaling in Diffuse Large B-Cell Lymphoma
Keisuke Kataoka, MD, PhD
Kyoto University
Kyoto, Japan
Novel Genetic Mechanism of Immune Evasion Involving PD-L1 in Aggressive Lymphomas
Marvyn Koning, BSc
Leiden University Medical Center
Leiden, Netherlands
Autonomous, Antigen-Independent B-Cell Receptor Signaling in Non-GCB DLBCL
Robert Kridel
BC Cancer Agency
Vancouver, Canada
Clonal Dynamics Shaping Histological Transformation and Progression in Follicular Lymphoma
Olga Kutovaya, PhD
BC Cancer Agency
Vancouver, Canada
The Role of UBR5 Mutations in the Pathogenesis of Mantle Cell Lymphoma
Anja Mottok, MD
BC Cancer Agency
Vancouver, Canada
Characterization of Genomic Rearrangements Involving CIITA and SOCS1 Using Targeted Capture Sequencing of Archival Tissue Specimens
Samuel Ng, MD, PhD
Dana-Farber Cancer Institute
Boston, MA
Exploring RhoA G17V-Mediated T-Cell Dysfunction
Georgios Pongas, MD
National Cancer Institute, National Institutes of Health
Bethesda, MD
Early Adaptive Responses of PI3K Activity Are Mediated Through PI3K-Alpha After PI3K-Delta Inhibition in ABC-DLBCL Cell Lines With MYD88 and CD79A or CD79B Mutations
Julie Ross, PhD
Institut de Recherches Cliniques de Montréal (IRCM)
Montreal, Canada
Loss of Functional Miz-1 Impairs c-Myc-Dependent B Cell Lymphomagenesis
Florian Scherer, MD
Stanford University Medical Center
Stanford, CA
Biopsy-Free Genotyping and Molecular Subtyping of Diffuse Large B-Cell Lymphoma
Lili Wang, MD, PhD
Harvard Medical School
Boston, MA
Integrated Dissection of Genetic and Transcriptional Heterogeneity in CLL by Single Cell Analysis

2014 Recipients

Wendy Béguelin, PhD
Weill Cornell Medical College
New York City, NY
EZH2 Cooperates With BCL6 to Induce Normal Germinal Center Reaction and Lymphomas Through Assembly of Non-Canonical PRC1 Complexes at Bivalent Chromatin Domains
Mariano Cardenas, PhD
Weill Medical College of Cornell University
New York, NY
Identification of Targetable Functional Groups on BCL6 Dimer Allows Rational Design of Potent and Selective Inhibitors With Activity in GCB- and ABC-Dlbcls
Katerina Hatzi, PhD
Weill Cornell Medical College
New York, NY
The Histone Demethylase LSD1 Acts as a BCL6 Corepressor in Germinal Center B Cells
Ondrej Havranek
The University of Texas MD Anderson Cancer Center
Houston, TX
The B-Cell Receptor Is Required for Optimal Viability, Growth, and Chemotherapy Resistance of Diffuse Large B-Cell Lymphoma Cell Lines of the Germinal Center B-Cell Subtype
Chuanxin Huang, PhD
Weill Cornell Medical College
New York, NY
The Bcl6 RD2 Domain Governs Commitment of Activated B-Cells to Form Germinal Centers
Yanwen Jiang, PhD
Weill Cornell Medical College
New York, NY
Deep-Sequencing Reveals Clonal Evolution Patterns and Mutation Events Associated With Relapse in B-Cell Lymphomas
Melanie Märklin, MSc
University of Tuebingen
Tuebingen, Germany
NFAT2 Is a Crucial Regulator of Anergy in CLL
Jonathan Reichel
Weill Cornell Medical College
New York, NY
Transcriptome Sequencing of the Reed-Sternberg Cells of Classical Hodgkin Lymphoma
Florian Schere, MD
University Medical Center Freiburg
Freiburg, Germany
Comprehensive Assessment of the Potential Role of B-Cell Receptor Antigen Recognition in the Pathogenesis of Follicular Lymphoma
Sally Trabucco
UMass Medical School
Worcester, MA
Inhibition of Bromodomain Proteins for the Treatment of Human Diffuse Large B-Cell Lymphoma
Jiyuan Zhang, PhD
Columbia University, Institute for Cancer Genetics
New York, NY
Inactivation of MLL2 Early During B Cell Differentiation Promotes Expansion of Mature Germinal Center B Cells: Implications for Lymphoma-Associated MLL2 Mutations